Hormone
A hormone is any member of a class of signaling molecules, produced by glands in multicellular organisms, that are transported by the circulatory system to target distant organs to regulate physiology and behavior. Hormones have diverse chemical structures, mainly of three classes:
- eicosanoids
- steroids
- amino acid/protein derivatives
Hormones serve to communicate between organs and tissues for physiological regulation and behavioral activities such as digestion, metabolism, respiration, tissue function, sensory perception, sleep, excretion, lactation, stress induction, growth and development, movement, reproduction, and mood manipulation. Hormones affect distant cells by binding to specific receptor proteins in the target cell, resulting in a change in cell function. When a hormone binds to the receptor, it results in the activation of a signal transduction pathway that typically activates gene transcription, resulting in increased expression of target proteins; non-genomic effects are more rapid, and can be synergistic with genomic effects. Amino acid–based hormones are water-soluble and act on the surface of target cells via second messengers; steroid hormones, being lipid-soluble, move through the plasma membranes of target cells to act within their nuclei.
Hormone secretion may occur in many tissues. Endocrine glands provide the cardinal example, but specialized cells in various other organs also secrete hormones. Hormone secretion occurs in response to specific biochemical signals from a wide range of regulatory systems. For instance, serum calcium concentration affects parathyroid hormone synthesis; blood sugar affects insulin synthesis; and because the outputs of the stomach and exocrine pancreas become the input of the small intestine, the small intestine secretes hormones to stimulate or inhibit the stomach and pancreas based on how busy it is. Regulation of hormone synthesis of gonadal hormones, adrenocortical hormones, and thyroid hormones often depends on complex sets of direct-influence and feedback interactions involving the hypothalamic-pituitary-adrenal, -gonadal, and -thyroid axes.
Upon secretion, certain hormones, including protein hormones and catecholamines, are water-soluble and are thus readily transported through the circulatory system. Other hormones, including steroid and thyroid hormones, are lipid-soluble; to achieve widespread distribution, these hormones must bond to carrier plasma glycoproteins to form ligand-protein complexes. Some hormones are completely active when released into the bloodstream, while others are prohormones that must be activated in specific cells through a series of activation steps that are commonly highly regulated. The endocrine system secretes hormones directly into the bloodstream, typically via fenestrated capillaries, whereas the exocrine system secretes its hormones indirectly using ducts. Hormones with paracrine function diffuse through the interstitial spaces to nearby target tissue.
Introduction and overview
Hormonal signaling involves the following steps:- Biosynthesis of a particular hormone in a particular tissue
- Storage and secretion of the hormone
- Transport of the hormone to the target cell
- Recognition of the hormone by an associated cell membrane or intracellular receptor protein
- Relay and amplification of the received hormonal signal via a signal transduction process: This then leads to a cellular response. The reaction of the target cells may then be recognized by the original hormone-producing cells, leading to a downregulation in hormone production. This is an example of a homeostatic negative feedback loop.
- Breakdown of the hormone.
Discovery
The discovery of hormones and endocrine signaling occurred during studies of how the digestive system regulates its activities, as explained at Secretin § Discovery.Arnold Adolph Berthold (1849)
was a German physiologist and zoologist, who, in 1849, had a question about the function of the testes. He noticed that in castrated roosters that they did not have the same sexual behaviors as roosters with their testes intact. He decided to run an experiment on male roosters to examine this phenomenon. He kept a group of roosters with their testes intact, and saw that they had normal sized wattles and combs, a normal crow, and normal sexual and aggressive behaviors. He also had a group with their testes surgically removed, and noticed that their secondary sexual organs were decreased in size, had a weak crow, did not have sexual attraction towards females, and were not aggressive. He realized that this organ was essential for these behaviors, but he did not know how. To test this further, he removed one testis and placed it in the abdominal cavity. The roosters acted and had normal physical anatomy. He was able to see that location of the testes do not matter. He then wanted to see if it was a genetic factor that was involved in the testes that provided these functions. He transplanted a testis from another rooster to a rooster with one testis removed, and saw that they had normal behavior and physical anatomy as well. Berthold determined that the location or genetic factors of the testes do not matter in relation to sexual organs and behaviors, but that some chemical in the testes being secreted is causing this phenomenon. It was later identified that this factor was the hormone testosterone.Bayliss and Starling (1902)
and Ernest Starling, a physiologist and biologist, respectively, wanted to see if the nervous system had an impact on the digestive system. They knew that the pancreas was involved in the secretion of digestive fluids after the passage of food from the stomach to the intestines, which they believed to be due to the nervous system. They cut the nerves to the pancreas in an animal model and discovered that it was not nerve impulses that controlled secretion from the pancreas. It was determined that a factor secreted from the intestines into the bloodstream was stimulating the pancreas to secrete digestive fluids. This factor was named secretin: a hormone, although the term hormone was not coined until 1905 by Starling.Types of signaling
Hormonal effects are dependent on where they are released, as they can be released in different manners. Not all hormones are released from a cell and into the blood until it binds to a receptor on a target. The major types of hormone signaling are:- Endocrine – Acts on the target cell after being released into the bloodstream.
- Paracrine – Acts on a nearby cell and does not have to enter general circulation.
- Autocrine – Affects the cell type that secreted it and causes a biological effect.
- Intracrine – Acts intracellularly on the cell that synthesized it.
Chemical classes
Vertebrate
;Peptide: Peptide hormones are made of a chain of amino acids that can range from just 3 to hundreds of amino acids. Examples include oxytocin and insulin. Their sequences are encoded in DNA and can be modified by alternative splicing and/or post-translational modification. They are packed in vesicles and are hydrophilic, meaning that they are soluble in water. Due to their hydrophilicity, they can only bind to receptors on the membrane, as travelling through the membrane is unlikely. However, some hormones can bind to intracellular receptors through an intracrine mechanism.;Amino acid: Amino acid hormones are derived from amino acid, most commonly tyrosine. They are stored in vesicles. Examples include melatonin and thyroxine.
;Eicosanoid: Eicosanoids hormones are derived from lipids such as arachidonic acid, lipoxins and prostaglandins. Examples include prostaglandin and thromboxane. These hormones are produced by cyclooxygenases and lipoxygenases. They are hydrophobic and act on membrane receptors.
;Steroid: Steroid hormones are derived from cholesterol. Examples include the sex hormones estradiol and testosterone as well as the stress hormone cortisol. Steroid contain four fused rings. They are lipophilic and hence can cross membranes to bind to intracellular nuclear receptors.
Invertebrate
Compared with vertebrates, insects and crustaceans possess a number of structurally unusual hormones such as the juvenile hormone, a sesquiterpenoid.Plant
Examples include abscisic acid, auxin, cytokinin, ethylene, and gibberellin.Receptors
Most hormones initiate a cellular response by initially binding to either cell membrane associated or intracellular receptors. A cell may have several different receptor types that recognize the same hormone but activate different signal transduction pathways, or a cell may have several different receptors that recognize different hormones and activate the same biochemical pathway.Receptors for most peptide as well as many eicosanoid hormones are embedded in the plasma membrane at the surface of the cell and the majority of these receptors belong to the G protein-coupled receptor class of seven alpha helix transmembrane proteins. The interaction of hormone and receptor typically triggers a cascade of secondary effects within the cytoplasm of the cell, described as signal transduction, often involving phosphorylation or dephosphorylation of various other cytoplasmic proteins, changes in ion channel permeability, or increased concentrations of intracellular molecules that may act as secondary messengers. Some protein hormones also interact with intracellular receptors located in the cytoplasm or nucleus by an intracrine mechanism.
For steroid or thyroid hormones, their receptors are located inside the cell within the cytoplasm of the target cell. These receptors belong to the nuclear receptor family of ligand-activated transcription factors. To bind their receptors, these hormones must first cross the cell membrane. They can do so because they are lipid-soluble. The combined hormone-receptor complex then moves across the nuclear membrane into the nucleus of the cell, where it binds to specific DNA sequences, regulating the expression of certain genes, and thereby increasing the levels of the proteins encoded by these genes. However, it has been shown that not all steroid receptors are located inside the cell. Some are associated with the plasma membrane.
Effects
Hormones have the following effects on the body:- stimulation or inhibition of growth
- wake-sleep cycle and other circadian rhythms
- mood swings
- induction or suppression of apoptosis
- activation or inhibition of the immune system
- regulation of metabolism
- preparation of the body for mating, fighting, fleeing, and other activity
- preparation of the body for a new phase of life, such as puberty, parenting, and menopause
- control of the reproductive cycle
- hunger cravings
Regulation
The rate of hormone biosynthesis and secretion is often regulated by a homeostatic negative feedback control mechanism. Such a mechanism depends on factors that influence the metabolism and excretion of hormones. Thus, higher hormone concentration alone cannot trigger the negative feedback mechanism. Negative feedback must be triggered by overproduction of an "effect" of the hormone.Hormone secretion can be stimulated and inhibited by:
- Other hormones
- Plasma concentrations of ions or nutrients, as well as binding globulins
- Neurons and mental activity
- Environmental changes, e.g., of light or temperature
To release active hormones quickly into the circulation, hormone biosynthetic cells may produce and store biologically inactive hormones in the form of pre- or prohormones. These can then be quickly converted into their active hormone form in response to a particular stimulus.
Eicosanoids are considered to act as local hormones. They are considered to be "local" because they possess specific effects on target cells close to their site of formation. They also have a rapid degradation cycle, making sure they do not reach distant sites within the body.
Hormones are also regulated by receptor agonists. Hormones are ligands, which are any kinds of molecules that produce a signal by binding to a receptor site on a protein. Hormone effects can be inhibited, thus regulated, by competing ligands that bind to the same target receptor as the hormone in question. When a competing ligand is bound to the receptor site, the hormone is unable to bind to that site and is unable to elicit a response from the target cell. These competing ligands are called antagonists of the hormone.
Therapeutic use
Many hormones and their structural and functional analogs are used as medication. The most commonly prescribed hormones are estrogens and progestogens, thyroxine and steroids. Insulin is used by many diabetics. Local preparations for use in otolaryngology often contain pharmacologic equivalents of adrenaline, while steroid and vitamin D creams are used extensively in dermatological practice.A "pharmacologic dose" or "supraphysiological dose" of a hormone is a medical usage referring to an amount of a hormone far greater than naturally occurs in a healthy body. The effects of pharmacologic doses of hormones may be different from responses to naturally occurring amounts and may be therapeutically useful, though not without potentially adverse side effects. An example is the ability of pharmacologic doses of glucocorticoids to suppress inflammation.
Hormone-behavior interactions
At the neurological level, behavior can be inferred based on: hormone concentrations; hormone-release patterns; the numbers and locations of hormone receptors; and the efficiency of hormone receptors for those involved in gene transcription. Not only do hormones influence behavior, but also behavior and the environment influence hormones. Thus, a feedback loop is formed. For example, behavior can affect hormones, which in turn can affect behavior, which in turn can affect hormones, and so on.Three broad stages of reasoning may be used when determining hormone-behavior interactions:
- The frequency of occurrence of a hormonally dependent behavior should correspond to that of its hormonal source
- A hormonally dependent behavior is not expected if the hormonal source is non-existent
- The reintroduction of a missing behaviorally dependent hormonal source is expected to bring back the absent behavior
Comparison with neurotransmitters
- A hormone can perform functions over a larger spatial and temporal scale than can a neurotransmitter.
- Hormonal signals can travel virtually anywhere in the circulatory system, whereas neural signals are restricted to pre-existing nerve tracts
- Assuming the travel distance is equivalent, neural signals can be transmitted much more quickly than can hormonal signals. Neural signals can be sent at speeds up to 100 meters per second.
- Neural signalling is an all-or-nothing action, whereas hormonal signalling is an action that can be continuously variable as dependent upon hormone concentration.